^Hsu SY، Lin P، Hsueh AJ (نوفمبر 1998). "BOD (Bcl-2-related ovarian death gene) is an ovarian BH3 domain-containing proapoptotic Bcl-2 protein capable of dimerization with diverse antiapoptotic Bcl-2 members". Mol Endocrinol. ج. 12 ع. 9: 1432–40. DOI:10.1210/mend.12.9.0166. PMID:9731710.
Wang K، Yin XM، Chao DT، وآخرون (1996). "BID: a novel BH3 domain-only death agonist". Genes Dev. ج. 10 ع. 22: 2859–69. DOI:10.1101/gad.10.22.2859. PMID:8918887.
Zha J، Harada H، Yang E، وآخرون (1997). "Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)". Cell. ج. 87 ع. 4: 619–28. DOI:10.1016/S0092-8674(00)81382-3. PMID:8929531.
Puthalakath H، Huang DC، O'Reilly LA، وآخرون (1999). "The proapoptotic activity of the Bcl-2 family member Bim is regulated by interaction with the dynein motor complex". Mol. Cell. ج. 3 ع. 3: 287–96. DOI:10.1016/S1097-2765(00)80456-6. PMID:10198631.
Ohi N، Tokunaga A، Tsunoda H، وآخرون (1999). "A novel adenovirus E1B19K-binding protein B5 inhibits apoptosis induced by Nip3 by forming a heterodimer through the C-terminal hydrophobic region". Cell Death Differ. ج. 6 ع. 4: 314–25. DOI:10.1038/sj.cdd.4400493. PMID:10381623.
Bouillet P، Metcalf D، Huang DC، وآخرون (1999). "Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity". Science. ج. 286 ع. 5445: 1735–8. DOI:10.1126/science.286.5445.1735. PMID:10576740.
Dijkers PF، Medema RH، Lammers JW، وآخرون (2000). "Expression of the pro-apoptotic Bcl-2 family member Bim is regulated by the forkhead transcription factor FKHR-L1". Curr. Biol. ج. 10 ع. 19: 1201–4. DOI:10.1016/S0960-9822(00)00728-4. PMID:11050388.
Bouillet P، Zhang LC، Huang DC، وآخرون (2001). "Gene structure alternative splicing, and chromosomal localization of pro-apoptotic Bcl-2 relative Bim". Mamm. Genome. ج. 12 ع. 2: 163–8. DOI:10.1007/s003350010242. PMID:11210187.
Putcha GV، Moulder KL، Golden JP، وآخرون (2001). "Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis". Neuron. ج. 29 ع. 3: 615–28. DOI:10.1016/S0896-6273(01)00238-0. PMID:11301022.
Murray S، Halford S، Ebenezer ND، وآخرون (2001). "Assignment of BCL2L11 to human chromosome band 2p13 with somatic cell and radiation hybrids". Cytogenet. Cell Genet. ج. 92 ع. 3–4: 353. DOI:10.1159/000056930. PMID:11435715.
Mami U، Miyashita T، Shikama Y، وآخرون (2002). "Molecular cloning and characterization of six novel isoforms of human Bim, a member of the proapoptotic Bcl-2 family". FEBS Lett. ج. 509 ع. 1: 135–41. DOI:10.1016/S0014-5793(01)03145-3. PMID:11734221.
Bouillet P، Purton JF، Godfrey DI، وآخرون (2002). "BH3-only Bcl-2 family member Bim is required for apoptosis of autoreactive thymocytes". Nature. ج. 415 ع. 6874: 922–6. DOI:10.1038/415922a. PMID:11859372.
Liu JW، Chandra D، Tang SH، وآخرون (2002). "Identification and characterization of Bimgamma, a novel proapoptotic BH3-only splice variant of Bim". Cancer Res. ج. 62 ع. 10: 2976–81. PMID:12019181.
Terradillos O، Montessuit S، Huang DC، Martinou JC (2002). "Direct addition of BimL to mitochondria does not lead to cytochrome c release". FEBS Lett. ج. 522 ع. 1–3: 29–34. DOI:10.1016/S0014-5793(02)02871-5. PMID:12095614.
Sugiyama T، Shimizu S، Matsuoka Y، وآخرون (2002). "Activation of mitochondrial voltage-dependent anion channel by apro-apoptotic BH3-only protein Bim". Oncogene. ج. 21 ع. 32: 4944–56. DOI:10.1038/sj.onc.1205621. PMID:12118373.